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MiNK Therapeutics, Inc. reports developments in clinical-stage biopharmaceutical programs built around allogeneic invariant natural killer T cell therapies. Its proprietary iNKT platform is designed to bridge innate and adaptive immunity, with agenT-797 as an off-the-shelf allogeneic iNKT cell therapy evaluated across cancer, immune-mediated disease and pulmonary immune-failure settings.
Company news commonly covers clinical and translational data, scientific-conference presentations, financial results, platform expansion, strategic collaborations and non-dilutive funding tied to iNKT cell therapy programs. Recurring program areas include solid tumors, gastroesophageal cancer, graft-versus-host disease, severe pulmonary inflammation, ARDS or hypoxemic pneumonia, and PRAME-targeted TCR-engineered iNKT approaches for pediatric cancers.
MiNK Therapeutics (NASDAQ: INKT), a clinical-stage biopharmaceutical company focused on developing innovative invariant natural killer T (iNKT) cell therapies, has scheduled its second quarter 2025 financial results announcement for August 14, 2025, before market opening.
The company will host a conference call and webcast at 8:30 a.m. ET to discuss the quarterly results and provide a corporate update. Investors can access the call through dial-in numbers for New York (646-307-1963) and USA/Canada (800-715-9871) using Conference ID 1149380, or via webcast on the company's investor relations website.
MiNK Therapeutics (NASDAQ: INKT) announced a publication in Frontiers in Immunology highlighting their innovative iNKT cell therapy platform. The article focuses on their lead program agenT-797, an unmodified allogeneic iNKT therapy, which has shown promising results in solid tumors without requiring lymphodepletion or genetic modification.
The company reported a notable case where a patient with metastatic testicular cancer achieved complete remission using agenT-797 combined with anti-PD-1 therapy, remaining disease-free for over two years. Additionally, in a Phase 2 trial for second-line gastric cancer, the therapy demonstrated enhanced tumor infiltration and durable disease control.
MiNK is also advancing MiNK-215, an IL-15-armored, FAP-targeting CAR-iNKT cell therapy designed to overcome stromal barriers in resistant tumors.
MiNK Therapeutics (NASDAQ: INKT) announced a groundbreaking publication in Nature's Oncogene detailing a complete remission case in metastatic testicular cancer using their agenT-797 allogeneic iNKT cell therapy. The patient, who had previously failed multiple treatments including platinum-based chemotherapy and immunotherapies, achieved complete remission lasting over two years after a single infusion of agenT-797 with nivolumab.
The therapy demonstrated remarkable safety with no cytokine release syndrome or graft-versus-host disease. Additionally, MiNK presented promising Phase 2 trial data in gastric cancer showing extended survival beyond 12 months in several patients, with a separate case reporting 42% tumor reduction and nine months progression-free survival.
MiNK Therapeutics (NASDAQ: INKT), a clinical-stage biopharmaceutical company focused on developing allogeneic, off-the-shelf, invariant natural killer T (iNKT) cell therapies, has scheduled its first quarter 2025 financial results release for May 15, 2025, before market opening. The company will host a conference call and webcast at 8:30 a.m. ET to discuss the results and provide a corporate update.
Investors can access the conference call via dial-in numbers 646-307-1963 (New York) or 800-715-9871 (USA & Canada) using Conference ID 9822477. A live webcast and replay will be available through the company's investor relations website.
MiNK Therapeutics (NASDAQ: INKT) reported its Q4 and full year 2024 financial results, highlighting progress in developing allogeneic iNKT cell therapies. The company's lead candidate, AgenT-797, showed promising results in cancer treatment and inflammatory lung conditions, with an 80% survival rate in VV ECMO patients versus 10% in hospital controls.
Key developments include a Phase 2 trial in advanced gastric cancer, next-generation PRAME-TCR iNKTs development, and MiNK-215 showing anti-tumor activity in colorectal cancer models. The company secured a research collaboration with Autonomous Therapeutics and raised $5.8M in private financing.
Financial results show a year-end cash balance of $4.6M, reduced cash burn with operations using $9.6M in 2024 compared to $15.8M in 2023. Net loss improved to $10.8M ($2.86 per share) in 2024 from $22.5M ($6.54 per share) in 2023.
MiNK Therapeutics (NASDAQ: INKT), a clinical-stage biopharmaceutical company focused on developing allogeneic, off-the-shelf, invariant natural killer T (iNKT) cell therapies, has scheduled its fourth quarter and full year 2024 financial results release for March 18th, 2025, before market opening.
The company will host a conference call and webcast at 8:30 a.m. ET on the same day, featuring a corporate update and discussion of financial results. Investors can access the call via phone at 646-307-1963 (New York) or 800-715-9871 (USA & Canada) using Conference ID 8023784, or through the company's investor relations website.
MiNK Therapeutics (NASDAQ: INKT) presented new translational data from its ongoing Phase 2 study of allo-iNKTs (agenT-797) at the AACR IO Annual Meeting. The study evaluates agenT-797 in combination with botensilimab and balstilimab (BOT/BAL) in patients with refractory gastroesophageal cancer.
The data demonstrated synergy between allo-iNKT cells, checkpoint inhibitors, and chemotherapy, showing robust immune reactivation in unresponsive tumors. Key findings include significant increases in interferon-gamma levels and enhanced tumor infiltration by T cells and antigen-presenting cells. The study revealed that optimal results were achieved when agenT-797 was administered concurrently with checkpoint inhibitors before chemotherapy.
MiNK's allogeneic manufacturing process can generate billions of donor-derived iNKT cells in a single run, producing thousands of doses for global distribution, potentially reducing costs and improving patient access.