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Tvardi Therapeutics (NASDAQ: TVRD) has completed enrollment in its Phase 2 clinical trial of TTI-101 for treating idiopathic pulmonary fibrosis (IPF). The REVERT IPF trial is a randomized, double-blind, placebo-controlled study evaluating TTI-101, an oral small molecule STAT3 inhibitor. The trial focuses on safety and lung function (FVC) endpoints, with topline data expected in Q4 2025.
TTI-101's unique dual mechanism of action inhibits STAT3-driven proliferation while activating T-cells, which has shown promise in preclinical studies by reducing lung fibrosis and restoring lung function. IPF is a chronic, progressive lung disease with poor prognosis, where current treatments only slow progression without halting or reversing fibrosis.
Tvardi Therapeutics (NASDAQ: TVRD), a clinical-stage biopharmaceutical company developing oral small molecule therapies targeting STAT3 for fibrosis-driven diseases, has announced its participation in two upcoming investor conferences. The company will join the Piper Sandler 3rd Annual Virtual Lung Symposium on May 27, 2025, at 11:30 AM ET for a fireside chat. Additionally, Tvardi will present at the Jefferies Global Healthcare Conference in New York on June 4, 2025, at 11:40 AM ET, which will include a corporate presentation and investor meetings. The Jefferies presentation will be available via webcast on Tvardi's Investors' website with a 60-day replay period.
Tvardi Therapeutics (NASDAQ: TVRD) announced its upcoming presentation at the American Thoracic Society 2025 Annual Conference in San Francisco. The presentation will highlight research on their STAT3 inhibitor, TTI-101, for treating idiopathic pulmonary fibrosis (IPF). Key findings show that ~60% of IPF-associated dysregulated genes are not addressed by current approved therapies (nintedanib and pirfenidone), termed the "IPF therapeutic gap." Research using single-cell RNA sequencing identified STAT3 as a dominant regulatory transcription factor in IPF. Notably, TTI-101 showed greater repression of genes within alveolar fibroblasts compared to existing treatments and demonstrated ability to downregulate genes involved in extracellular matrix production, including collagen genes.
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