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Disc Medicine Presents Initial Results from RESTORE-PV Phase 2 Trial in Patients with Polycythemia Vera (PV) at the 14th Society of Hematologic Oncology (SOHO) Annual Meeting

Early Phase 2 RESTORE-PV data show DISC-3405 reduced phlebotomy needs and maintained hematocrit control in treated polycythemia vera patients.

(Moderate)
(Positive)

Disc Medicine (IRON) reported initial Phase 2 RESTORE-PV results for DISC-3405 in polycythemia vera at the 2026 SOHO meeting.

In Cohort A, treatment increased hepcidin, lowered serum iron and raised ferritin, with mean hematocrit stably maintained below 45% through week 26 and associated initial symptom improvement. Among 13 patients completing 26 weeks, mean phlebotomy events fell from 4.0 in the 26 weeks before Day 1 to 0.6 in the 26 weeks after (p<0.0001); 61.5% remained phlebotomy-free through 26 weeks, and 77.8% of nine patients completing weeks 12–32 were phlebotomy-free in that period. DISC-3405 was generally well tolerated with mostly disease-consistent adverse events and mild, self-limited injection site reactions.

The company plans further RESTORE-PV and sickle cell disease updates and is preparing selcodebart (DISC-0974) for potential pivotal development in myelofibrosis anemia.

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Positive

  • Mean phlebotomies dropped from 4.0 to 0.6 over 26 weeks (n=13)
  • 61.5% of RESTORE-PV Cohort A completers remained phlebotomy-free through 26 weeks
  • 77.8% of patients completing weeks 12–32 stayed phlebotomy-free in that interval
  • Mean hematocrit was maintained stably below 45% through week 26
  • DISC-3405 showed dose-proportional pharmacokinetics and on-target iron modulation
  • Safety profile was generally well tolerated with mild injection reactions
  • Two programs, DISC-3405 and selcodebart, are advancing toward potential pivotal trials
  • Company guides to additional RESTORE-PV and sickle cell data by end of 2026

Negative

  • Efficacy data currently available only for Cohort A, not full enrollment
  • RESTORE-PV results are initial Phase 2 data with small analyzed sample (n=13)

News Explained

The disclosure is an interim Phase 2 readout: efficacy results cover Cohort A, where 13 participants completed 26 weeks, while Cohort B had 18 of 20 participants dosed but only baseline and safety data were presented, so the trial’s clinical evidence remains incomplete.

Market Context

At publication, IRON was down 3.02% on pre-headline data, while the momentum scanner showed only one...
Analysis

At publication, IRON was down 3.02% on pre-headline data, while the momentum scanner showed only one peer moving up; the RESTORE-PV results arrived without a recorded two-peer same-direction sector signal.

Key Figures

Trial enrollment: 40 participants Dose: 300 mg Q2 weeks or Q4 weeks Study duration: 26 weeks +5 more
Trial enrollment
40 participants
RESTORE-PV Phase 2 trial
Dose
300 mg Q2 weeks or Q4 weeks
Cohort A or Cohort B
Study duration
26 weeks
Cohort A efficacy assessment
Hematocrit
<45%
Mean hematocrit maintained through week 26
Mean phlebotomy events
4.0 to 0.6
26 weeks at baseline versus 26 weeks post-Day 1; n=13
Phlebotomy-free participants
61.5%
Through 26 weeks post-baseline
Maintenance-period phlebotomy-free participants
77.8%
Weeks 12-32; n=9
P-value
p<0.0001
Reduction in mean total phlebotomy events

Key Terms

polycythemia vera, hepcidin, tmprss6, pharmacodynamics, +1 more
5 terms
polycythemia vera medical
"patients with polycythemia vera (PV)"
A rare, long-term blood disorder in which the body makes too many red blood cells, thickening the blood and raising the risk of clots, bleeding, fatigue and other complications. Think of it like a faucet left partially open that slowly overfills a sink — the excess cells create strain and danger over time. Investors care because the condition drives demand for diagnostics, treatments and ongoing care, influences clinical trial and regulatory outcomes, and can affect revenue and costs for drugmakers, hospitals and insurers.
hepcidin medical
"increased hepcidin and lowered serum iron"
Hepcidin is a small hormone produced by the liver that acts like a thermostat for the body's iron supply, telling the gut and storage sites when to absorb, release or hold onto iron. Investors watch hepcidin because drugs, tests or diagnostics that change its levels can treat common conditions such as anemia or iron overload, making them potential revenue drivers and regulatory milestones in healthcare markets.
tmprss6 medical
"targeting TMPRSS6 that has demonstrated phlebotomy reduction"
TMPRSS6 is a human gene that makes a liver enzyme (matriptase-2) acting like a thermostat for the body’s iron levels by controlling production of the hormone hepcidin, which dictates how much iron is absorbed and stored. It matters to investors because changes or drugs that target this gene can alter iron-related diseases, create diagnostic markers, or enable new therapies for anemia or iron overload—outcomes that affect the commercial value of biomedical products and companies.
pharmacodynamics medical
"the established pharmacodynamics of DISC-3405"
Pharmacodynamics is how a drug actually affects the body — the strength, type and duration of its effects and the relationship between dose and response. Think of it like how turning a thermostat changes room temperature: it shows what the drug does and how much is needed to get the desired effect. Investors care because these properties drive clinical success, dosing convenience, safety profile and competitive advantage, all of which influence commercial potential and regulatory approval.
monoclonal antibody medical
"DISC-3405 is an investigational, anti-TMPRSS6 monoclonal antibody"
A monoclonal antibody is a laboratory-made protein designed to recognize and attach to a specific target in the body, such as a disease-causing substance or cell. It functions like a highly precise lock-and-key tool, helping to treat or detect illnesses. For investors, companies developing monoclonal antibodies can represent promising opportunities in the healthcare sector, especially as these treatments often address unmet medical needs.

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  • Initial results from RESTORE-PV show DISC-3405 increased hepcidin and lowered serum iron, translating to controlled hematocrit, reduced phlebotomy, and improved symptoms in PV patients
  • DISC-3405 is the first monoclonal antibody targeting TMPRSS6 that has demonstrated phlebotomy reduction in patients with PV
  • Disc also presented an encore of the positive results from the Phase 2 RALLY-MF trial of selcodebart (DISC-0974) in patients with anemia of myelofibrosis (MF) and a new systematic literature review characterizing the burden of anemia associated with MF

WATERTOWN, Mass., Sept. 09, 2026 (GLOBE NEWSWIRE) -- Disc Medicine, Inc. (NASDAQ:IRON), a clinical-stage biopharmaceutical company focused on the discovery, development, and commercialization of novel treatments for patients suffering from serious hematologic diseases, today presented positive initial results from the RESTORE-PV Phase 2 trial of DISC-3405 in patients with polycythemia vera (PV). The data, presented in a poster session at the 2026 Society of Hematologic Oncology (SOHO) annual meeting in Houston, TX, demonstrated that treatment with DISC-3405 increased hepcidin and lowered serum iron, translating to reduction in phlebotomy, controlled hematocrit, and improved symptom burden in patients with PV. These data will be featured in an oral presentation at the SOHO conference tomorrow, September 10. Disc also presented an encore of the positive results from the Phase 2 RALLY-MF trial of selcodebart (DISC-0974) in patients with anemia of myelofibrosis (MF) and a new systematic literature review characterizing the humanistic burden of anemia associated with MF through patient reported outcomes.

“This first look at data from the RESTORE-PV trial shows that the established pharmacodynamics of DISC-3405 are translating well on important clinical measures,” said John Quisel, JD, PhD, President and Chief Executive Officer of Disc Medicine. “Iron restriction is proving to be a transformative treatment approach for a large population of patients with polycythemia vera, with the potential to address their crucial need for hematocrit control while managing symptom burden and reducing reliance on phlebotomy. Our goal for DISC-3405 is to deliver an optimal product presentation combining durable disease control with straightforward, controllable, and convenient dosing using a novel antibody approach. Between this update in PV and our earlier progress in MF this year, we have strengthened our commitment to hematologic oncology and now have two programs advancing toward potential pivotal development in myeloproliferative neoplasms.”

The Phase 2 multi-center, open-label RESTORE-PV trial enrolled 40 adult participants with PV, including 20 participants in Cohort A and 20 participants in Cohort B. In the trial, following a 4- to 12-week observation, participants undergo a 12-week dose escalation, then receive DISC-3405 subcutaneously at 300 mg Q2 weeks (Cohort A) or Q4 weeks (Cohort B) for 20 weeks, followed by up to 20 additional weeks of treatment at these respective doses. At the time of the data cut, all 20 participants in Cohort A were dosed and n=13 patients completed 26 weeks of the study, and 18 of 20 participants in Cohort B were dosed. Efficacy data was presented for Cohort A and baseline and safety data were presented for Cohorts A and B. Across escalation and maintenance periods, results demonstrated:

  • Dose-proportional PK, elevation of hepcidin, reduction of serum iron, and increase in ferritin (Cohort A)
  • Control of hematocrit, with mean hematocrit maintained stably <45% through week 26 which led to initial improvement in symptom burden (Cohort A)
  • DISC-3405 significantly reduced phlebotomy events in Cohort A participants. For n=13 patients completing 26 weeks of study:
    • Mean total phlebotomy events significantly decreased from 4.0 in 26 weeks at baseline to 0.6 in 26 weeks post-Day 1 (p<0.0001)
    • 61.5% of participants remained entirely phlebotomy-free post-baseline through 26 weeks
    • Of those who completed the first maintenance period (weeks 12-32, n=9), 77.8% of participants remained phlebotomy free during this period
  • DISC-3405 was generally well-tolerated with adverse events that are consistent with underlying disease and a low rate of injection site reactions which were mild and self-limited (Cohorts A and B)

Disc plans to provide an update on RESTORE-PV, as well as initial data from the Phase 1b trial of DISC-3405 in sickle cell disease, by the end of 2026.

With respect to selcodebart, the company also expects to share feedback from an end of phase 2 meeting with the U.S. Food and Drug Administration (FDA) and plans for pivotal development in anemia of MF by the end of the year.

About DISC-3405

DISC-3405 is an investigational, anti-TMPRSS6 (Transmembrane Serine Protease 6, also known as Matriptase-2) monoclonal antibody designed to increase hepcidin production and suppress serum iron. Disc in-licensed DISC-3405 from Mabwell Therapeutics in January 2023. The therapeutic potential for hepcidin induction includes treatment of diseases associated with iron overload, diseases of excess red blood cell production, or diseases otherwise enabled by iron availability. Disc has established clinical proof-of-mechanism of DISC-3405 in a Phase 1 study in healthy volunteers and initiated a Phase 2 trial in patients with polycythemia vera and a Phase 1b trial in patients with sickle cell disease.

DISC-3405 is an investigational agent and is not approved for use as a therapy in any jurisdiction worldwide.

About Polycythemia Vera

Polycythemia vera (PV) is a chronic and rare myeloproliferative neoplasm characterized by the abnormal proliferation of red blood cells. PV affects approximately 150,000 patients in the U.S. and has a similar prevalence in Europe. The overproduction of red blood cells alters the viscosity of blood, causing it to thicken and placing patients at an elevated risk of cardiovascular and thromboembolic events, such as heart attack and stroke. Patients also experience complications such as enlarged spleen and symptoms of their disease such as fatigue, pruritis, difficulty concentrating and others. Current therapy involves phlebotomy to physically remove blood and iron to limit erythropoiesis or treatment with cytoreductive agents, with the goal of reducing red blood cell count and managing symptoms.

About Disc Medicine

Disc Medicine is a clinical-stage biopharmaceutical company committed to discovering, developing, and commercializing novel treatments for patients who suffer from serious hematologic diseases. We are building a portfolio of innovative, potentially first-in-class therapeutic candidates that aim to address a wide spectrum of hematologic diseases by targeting fundamental biological pathways of red blood cell biology, specifically heme biosynthesis and iron homeostasis. For more information, please visit www.discmedicine.com.

Disc Cautionary Statement Regarding Forward-Looking Statements

This press release contains “forward-looking statements” within the meaning of the Private Securities Litigation Reform Act of 1995, including, but not limited to, express or implied statements regarding Disc’s expectations with respect to the next stages of its development programs for selcodebart and DISC-3405, including projected timelines for the initiation and completion of its clinical trials, anticipated timing of release of data, other clinical activities, and anticipated discussions with regulatory agencies. The use of words such as, but not limited to, “believe,” “expect,” “estimate,” “project,” “intend,” “future,” “potential,” “continue,” “may,” “might,” “plan,” “will,” “should,” “seek,” “anticipate,” or “could” or the negative of these terms and other similar words or expressions that are intended to identify forward-looking statements. Forward-looking statements are neither historical facts nor assurances of future performance. Instead, they are based on Disc’s current beliefs, expectations and assumptions regarding the future of Disc’s business, future plans and strategies, clinical results and other future conditions. New risks and uncertainties may emerge from time to time, and it is not possible to predict all risks and uncertainties. No representations or warranties (expressed or implied) are made about the accuracy of any such forward-looking statements.

Disc may not actually achieve the plans, intentions or expectations disclosed in these forward-looking statements, and investors should not place undue reliance on these forward-looking statements. Actual results or events could differ materially from the plans, intentions and expectations disclosed in the forward-looking statements as a result of a number of material risks and uncertainties including but not limited to: the adequacy of Disc’s capital to support its future operations and its ability to successfully initiate and complete clinical trials; the nature, strategy and focus of Disc; the difficulty in predicting the time and cost of development of Disc’s product candidates; Disc’s plans to research, develop and commercialize its current and future product candidates; the timing of initiation of Disc’s planned preclinical studies and clinical trials; the timing of the availability of data from Disc’s clinical trials; Disc’s ability to identify additional product candidates with significant commercial potential and to expand its pipeline in hematological diseases; the timing and anticipated results of Disc’s preclinical studies and clinical trials and the risk that the results of Disc’s preclinical studies and clinical trials may not be predictive of future results in connection with future studies or clinical trials and may not support further development and marketing approval; and the other risks and uncertainties described in Disc’s filings with the Securities and Exchange Commission, including in the “Risk Factors” section of Disc’s Annual Report on Form 10-K for the year ended December 31, 2025, and in subsequent Quarterly Reports on Form 10-Q. Any forward-looking statement speaks only as of the date on which it was made. None of Disc, nor its affiliates, advisors or representatives, undertake any obligation to publicly update or revise any forward-looking statement, whether as result of new information, future events or otherwise, except as required by law.

Media Contact

Peg Rusconi
Deerfield Group
peg.rusconi@deerfieldgroup.com

Investor Relations Contact

Christina Tartaglia
Precision AQ
christina.tartaglia@precisionaq.com


FAQ

How is the RESTORE-PV Phase 2 trial of DISC-3405 designed?

RESTORE-PV is a multi-center, open-label Phase 2 trial enrolling 40 adults with polycythemia vera, split into 20 participants in Cohort A and 20 in Cohort B. After a 4–12 week observation period, participants undergo a 12-week dose escalation, then receive subcutaneous DISC-3405 at 300 mg every 2 weeks in Cohort A or every 4 weeks in Cohort B for 20 weeks, followed by up to 20 additional weeks at these maintenance doses.

How many RESTORE-PV patients had been treated at the data cut?

At the time of the data cut, all 20 participants in Cohort A had been dosed and 13 had completed 26 weeks of the study, while 18 of 20 participants in Cohort B had been dosed. Efficacy data were presented for Cohort A, with baseline and safety data for both cohorts.

What were the key pharmacodynamic findings for DISC-3405 in PV?

Across dose escalation and maintenance in Cohort A, DISC-3405 showed dose-proportional pharmacokinetics, increased hepcidin, reduced serum iron, and increased ferritin. The company stated that these changes translated into controlled hematocrit and initial improvement in symptom burden.

What is DISC-3405 and which indications is it being studied in?

DISC-3405 is an investigational anti-TMPRSS6 monoclonal antibody designed to increase hepcidin and suppress serum iron. It is being studied in a Phase 2 trial in polycythemia vera and a Phase 1b trial in sickle cell disease, following earlier proof-of-mechanism in a Phase 1 healthy volunteer study.

What development milestones did Disc outline for 2026?

Disc plans to provide an update on RESTORE-PV and initial data from the Phase 1b trial of DISC-3405 in sickle cell disease by the end of 2026. For selcodebart (DISC-0974) in anemia of myelofibrosis, the company expects feedback from an end of Phase 2 FDA meeting and to share its plans for pivotal development by year-end.

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