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Telomir Pharmaceuticals Announces Peer-Reviewed Publication in Biomedicine & Pharmacotherapy Demonstrating Restoration of Insulin Sensitivity with Telomir-Zn in a Preclinical Type 2 Diabetes Model

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Telomir Pharmaceuticals (NASDAQ:TELO) reported a peer-reviewed publication in Biomedicine & Pharmacotherapy showing Telomir‑Zn restored insulin sensitivity and improved glucose homeostasis in a diet-induced zebrafish Type 2 diabetes model.

After 14 days, HOMA-IR fell from about 10–12 to ~3, with dose-dependent effects, supporting Telomir‑Zn's metal‑homeostasis mechanism and its FDA‑cleared Phase 1/2 TNBC program.

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AI-generated analysis. How Rhea-AI works. Not financial advice.

Positive

  • HOMA-IR reduced from approximately 10–12 to ~3 after 14 days of Telomir‑Zn
  • Dose-dependent improvements in fasting glucose, glucose tolerance and insulin levels
  • Peer-reviewed publication in Biomedicine & Pharmacotherapy adds independent validation
  • Findings support Telomir‑Zn’s metal-homeostasis mechanism across multiple disease models
  • Telomir‑Zn has FDA-cleared IND for Phase 1/2 trial TELO-001 in TNBC
  • Second peer-reviewed Telomir‑Zn publication in 2026 strengthens scientific dossier

Negative

  • Data are limited to a preclinical diet-induced zebrafish Type 2 diabetes model
  • No human clinical data for metabolic disease with Telomir‑Zn are reported

News Market Reaction – TELO

+2.38%
+2.38% News Effect

On the day this news was published, TELO gained 2.38%, reflecting a moderate positive market reaction.

Data tracked by StockTitan Argus on the day of publication.

What This Means

This announcement adds preclinical Type 2 diabetes data to Telomir‑Zn’s growing body of peer‑reviewe...
Analysis

This announcement adds preclinical Type 2 diabetes data to Telomir‑Zn’s growing body of peer‑reviewed support, showing HOMA‑IR improvement from about 10–12 to 3 after 14 days of treatment. Together with prior Wilson’s disease and TNBC findings, it reinforces the metal‑homeostasis mechanism across distinct indications. Investors may track upcoming TELO‑001 Phase 1/2 progress, additional publications, and future SEC filings for clarity on funding and development timelines.

Key Figures

Treatment duration: 14 days Baseline HOMA-IR: 10–12 Post-treatment HOMA-IR: 3 +4 more
7 metrics
Treatment duration 14 days Telomir‑Zn treatment period in Type 2 diabetes zebrafish study
Baseline HOMA-IR 10–12 Untreated diabetic zebrafish prior to Telomir‑Zn
Post-treatment HOMA-IR 3 After 14 days of Telomir‑Zn treatment
Assessment days Day 7 and Day 14 Timepoints where metabolic improvements were consistent
Trial phase Phase 1/2 Planned TELO‑001 clinical trial in TNBC
Model type Diet-induced zebrafish T2DM model Preclinical Type 2 diabetes mellitus study setting
Peer-reviewed publications 2026 Second publication Second Telomir‑Zn peer-reviewed paper in 2026

Historical Context

5 past events · Latest: May 20 (Positive)
Pattern 5 events
Date Event Sentiment 24h Move Catalyst
May 20 Preclinical data – Wilson’s Positive +4.5% Wilson’s disease zebrafish data showed multi-endpoint benefits and improved survival.
Apr 30 IND clearance TNBC Positive -3.5% FDA cleared IND for Telomir‑Zn Phase 1/2 trial in advanced TNBC.
Apr 24 Acquisition completed Positive +1.4% Closed TELI acquisition, consolidating global Telomir‑Zn rights and funding milestones.
Mar 31 IND submitted TNBC Positive +14.0% Submitted IND for Telomir‑Zn with supportive preclinical efficacy and safety data.
Feb 17 Preclinical TNBC data Positive +5.5% Reported iron‑dependent TNBC cell mortality across multiple in vitro models.

24h Move is the share-price change in the day after each event; other market factors may also have contributed.

Pattern Detected

Most prior positive Telomir-Zn milestones led to aligned price gains, with one notable divergence on FDA IND clearance.

Recent Company History

Over the last few months, Telomir reported multiple Telomir‑Zn milestones, including broad TNBC cell mortality on Feb 17, 2026, IND submission on Mar 31, 2026, and FDA IND clearance on Apr 30, 2026. It then completed the TELI acquisition on Apr 24, 2026 and added Wilson’s disease preclinical data on May 20, 2026. Today’s Type 2 diabetes zebrafish data extend this mechanistic story into metabolic disease models.

Key Terms

homa-ir, oral glucose tolerance testing (ogtt), oxidative stress, mitochondrial dysfunction, +4 more
8 terms
homa-ir medical
"HOMA-IR values of approximately 10-12, consistent with severe insulin resistance."
HOMA-IR is a calculated score that estimates how resistant a person’s cells are to insulin, using routine fasting blood glucose and insulin measurements. Investors track changes in HOMA-IR in clinical data because it acts like a simple gauge of metabolic effect: improvements suggest a therapy could meaningfully treat or prevent diabetes-related conditions, which informs commercial potential, regulatory outlook and market valuation.
oral glucose tolerance testing (ogtt) medical
"Improved glucose clearance during oral glucose tolerance testing (OGTT)"
A test that measures how a person’s blood sugar responds after drinking a measured glucose solution; blood samples are taken before and at intervals after the drink to see how quickly the body clears the sugar. Investors care because OGTT results are used in clinical trials, diagnostic approvals, and screening guidelines, which influence demand for diabetes drugs, medical devices, and lab services—think of it as a stress test that reveals market needs in diabetes care.
oxidative stress medical
"associated with oxidative stress, mitochondrial dysfunction, and metabolic dysregulation."
Oxidative stress is a biological imbalance where damaging, unstable molecules overwhelm the body’s neutralizing defenses, similar to how rust forms when metal is exposed to oxygen and moisture. Investors should care because oxidative stress is linked to many diseases and aging processes, influencing demand for drugs, diagnostics, supplements, and healthcare spending, and it can affect the commercial value and regulatory outlook of related products.
mitochondrial dysfunction medical
"associated with oxidative stress, mitochondrial dysfunction, and metabolic dysregulation."
Mitochondrial dysfunction is when the cell’s energy factories, called mitochondria, fail to make enough energy for cells to work properly, which can cause tissue damage and contribute to many diseases. For investors, it matters because therapies, tests, or devices aimed at fixing or detecting this problem can create market opportunities or clinical risk; think of it as a failing car battery that drives demand for repairs, replacements, and related medical products.
metal homeostasis medical
"mechanism of action through modulation of intracellular metal homeostasis."
Metal homeostasis is the biological process that keeps levels of metal ions—like iron, copper, and zinc—within a healthy range so cells can function properly, similar to a thermostat keeping a room at the right temperature. For investors, disruptions or therapies that alter metal balance matter because they can drive drug discovery, influence safety and toxicity profiles, affect diagnostic biomarkers, and shape regulatory decisions that impact company value.
epigenetic dysregulation medical
"pathways have been implicated in epigenetic dysregulation, treatment resistance,"
Epigenetic dysregulation is when the chemical tags and switches that control whether genes are turned on or off are disturbed, like a dimmer switch stuck at the wrong setting so instructions in the DNA are read incorrectly. For investors, it matters because such disturbances underlie many diseases and can create opportunities or risks for drug developers, diagnostics firms, and regulators: therapies that fix or detect these changes can become valuable assets, while unexpected safety or efficacy issues can affect company valuations.
investigational new drug (ind) regulatory
"received Investigational New Drug (IND) clearance from the U.S. Food and Drug Administration"
An investigational new drug (IND) is a drug or biologic that is being tested but has not yet been approved for general use; it is the application and formal status that allows a company to begin human clinical trials under regulator oversight. Investors care because an IND marks the transition from lab work to human testing — like getting a permit to run real-world experiments — which creates important milestones, costs, timelines and regulatory risk that drive a development-stage company's value.
triple-negative breast cancer medical
"Phase 1/2 clinical trial (TELO-001) in patients with advanced or metastatic triple-negative breast cancer."
Triple-negative breast cancer is a type of breast cancer that lacks three common markers used to identify and treat the disease effectively. Because it doesn’t respond to some targeted therapies, it can be more difficult to treat and may have a more aggressive progression. This impacts the development of new treatments and can influence the outlook for healthcare companies involved in cancer research and pharmaceuticals.

AI-generated analysis. How Rhea-AI works. Not financial advice.

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Insulin Resistance Was Normalized Following 14 Days of Treatment, Supporting Restoration of Glucose Metabolism and Insulin Sensitivity and Further Validating Telomir-Zn's Metal-Homeostasis Mechanism Across Multiple Disease Models.

MIAMI, FL / ACCESS Newswire / June 15, 2026 / Telomir Pharmaceuticals, Inc. (NASDAQ:TELO) ("Telomir" or the "Company"), a clinical-stage biotechnology company developing small-molecule therapeutics targeting epigenetic and metabolic drivers of cancer and age-related disease, today announced the peer-reviewed publication of preclinical data demonstrating restoration of insulin sensitivity and significant improvement in glucose homeostasis with Telomir-Zn in a diet-induced zebrafish model of Type 2 diabetes mellitus (T2DM).

The manuscript, titled "Telomir-Zn Restores Glucose Homeostasis and Reduces Insulin Resistance in a Diet-Induced Zebrafish Model of Type 2 Diabetes," has been published in Biomedicine & Pharmacotherapy.

The publication is available online in Biomedicine & Pharmacotherapy.

Publication Highlights

Type 2 diabetes mellitus is characterized by chronic hyperglycemia, impaired glucose utilization, and insulin resistance. Insulin resistance is a central driver of disease progression and is associated with oxidative stress, mitochondrial dysfunction, and metabolic dysregulation.

In the study, zebrafish fed a high-calorie diet developed significant metabolic dysfunction, including fasting hyperglycemia, hyperinsulinemia, impaired glucose tolerance, and HOMA-IR values of approximately 10-12, consistent with severe insulin resistance.

According to the publication, treatment with Telomir-Zn produced dose-dependent improvements across multiple independent measures of metabolic health, including:

  • Significant reduction in fasting blood glucose levels to near control levels

  • Improved glucose clearance during oral glucose tolerance testing (OGTT)

  • Reduction in fasting insulin concentrations

  • Significant improvement in HOMA-IR, a widely used measure of insulin resistance

Most notably, HOMA-IR values declined from approximately 10-12 in untreated diabetic animals to approximately 3 following 14 days of treatment, representing a substantial reversal of insulin resistance. Improvements were observed in a dose-dependent manner and remained consistent across both Day 7 and Day 14 assessments.

The authors concluded that Telomir-Zn restored key metabolic parameters associated with insulin resistance and glucose dysregulation, supporting further investigation of metal-modulating small molecules as a novel therapeutic approach for metabolic disease.

Mechanistic Relevance to Telomir-Zn's Clinical Development Program

The publication adds to a growing body of peer-reviewed evidence supporting Telomir-Zn's mechanism of action through modulation of intracellular metal homeostasis.

Accumulating scientific evidence suggests that dysregulation of intracellular iron and zinc balance contributes to oxidative stress, mitochondrial dysfunction, and altered cellular signaling across multiple disease states. In metabolic disease, these processes can impair insulin receptor signaling and glucose homeostasis. In cancer, similar pathways have been implicated in epigenetic dysregulation, treatment resistance, tumor progression, and aggressive disease biology.

Telomir-Zn was designed to modulate intracellular metal homeostasis and influence iron-dependent biological pathways through a zinc-coordinated small-molecule platform.

While Type 2 diabetes and triple-negative breast cancer represent distinct disease states, both involve oxidative stress, dysregulated metal homeostasis, and iron-dependent cellular signaling pathways targeted by Telomir-Zn.

The Company believes these findings provide additional independent, peer-reviewed validation of biological pathways underlying its lead clinical program. Telomir-Zn recently received Investigational New Drug (IND) clearance from the U.S. Food and Drug Administration (FDA) for the Company's Phase 1/2 clinical trial (TELO-001) in patients with advanced or metastatic triple-negative breast cancer.

Management Commentary

"What makes these findings particularly interesting is that excess intracellular iron has been implicated in both insulin resistance and cancer biology," said Dr. Itzchak Angel, Chief Scientific Advisor of Telomir Pharmaceuticals and corresponding author of the publication.

"In diabetes, iron-driven oxidative stress can impair insulin signaling and glucose metabolism. In cancer, iron-dependent pathways can contribute to epigenetic dysregulation and tumor progression. The ability of Telomir-Zn to produce meaningful biological effects in multiple disease models further strengthens our confidence that modulation of intracellular metal homeostasis may represent an important therapeutic strategy."

"This publication represents our second peer-reviewed publication on Telomir-Zn in 2026, supporting the biological mechanism underlying Telomir-Zn," said Erez Aminov, Chairman and Chief Executive Officer of Telomir Pharmaceuticals.

"Combined with our recently published Wilson's disease findings and our FDA-cleared Phase 1/2 TNBC program, we continue to build a growing body of scientific evidence supporting the broader potential of our metal-homeostasis platform. We now have an FDA-cleared IND, preparations underway for our Phase 1/2 TNBC study, and multiple peer-reviewed publications supporting our underlying biology. Our focus remains on execution as we advance Telomir-Zn into human clinical trials."

About Telomir Pharmaceuticals

Telomir Pharmaceuticals, Inc. (NASDAQ:TELO) is a clinical-stage biotechnology company developing small-molecule therapeutics targeting epigenetic and metabolic pathways implicated in cancer, aging, and degenerative disease. The Company's lead program, Telomir-Zn, is designed to modulate intracellular metal homeostasis and epigenetic regulation and has received IND clearance from the U.S. Food and Drug Administration for a Phase 1/2 clinical trial in Triple-Negative Breast Cancer. For more information, please visit https://telomirpharma.com/.

Forward-Looking Statements

This press release contains "forward-looking statements" within the meaning of the Private Securities Litigation Reform Act of 1995. These forward-looking statements generally can be identified by the use of words such as "anticipate," "expect," "plan," "can," "could," "would," "may," "will," "believe," "estimate," "forecast," "goal," "project," "guidance," "potential," "intend," "seek," "target" and other words of similar meaning, although not all forward-looking statements include these words.

Forward-looking statements may include, but are not limited to, statements regarding the therapeutic potential, mechanism of action, development plans, regulatory pathway, safety profile, clinical utility, market opportunity, and future development of Telomir-1 (Telomir-Zn) and the Company's other product candidates. Forward-looking statements may also include statements regarding the significance of the published preclinical findings, the relevance of such findings to the Company's oncology development programs, the advancement of the Company's Phase 1/2 TNBC clinical trial, and the potential applicability of Telomir-Zn across multiple disease areas.

These forward-looking statements are based on current expectations, estimates, forecasts, and projections, as well as management's beliefs and assumptions, and are subject to significant risks and uncertainties that could cause actual results to differ materially from those expressed or implied by such statements. These risks and uncertainties include, among others, risks related to preclinical and clinical development, the ability to obtain regulatory approvals, the outcome of future studies, reliance on third parties, intellectual property protection, financing needs, market conditions, and the other risks identified under the heading "Risk Factors" contained in the Company's Annual Report on Form 10-K and the Company's other filings with the U.S. Securities and Exchange Commission ("SEC").

Forward-looking statements contained in this press release speak only as of the date hereof, and the Company undertakes no obligation to update or revise such statements, whether as a result of new information, future events, or otherwise, except as required by applicable law.

We caution investors not to place undue reliance on the forward-looking statements contained in this press release. You are encouraged to read our filings with the SEC, available at the SEC website and in the "Investors" section of our website, for a discussion of these and other risks and uncertainties.

Contact Information

Krystina Quintana
Email: info@telomirpharma.com
Phone: (786) 396-6723

SOURCE: Telomir Pharmaceuticals, Inc



View the original press release on ACCESS Newswire

FAQ

What did Telomir Pharmaceuticals (NASDAQ:TELO) announce on June 15, 2026 about Telomir-Zn?

Telomir Pharmaceuticals announced a peer-reviewed publication showing Telomir‑Zn restored insulin sensitivity in a zebrafish Type 2 diabetes model. According to the company, treatment improved fasting glucose, glucose tolerance, fasting insulin and HOMA-IR, supporting its metal-homeostasis mechanism and broader development strategy.

How did Telomir-Zn affect HOMA-IR and insulin resistance in the zebrafish Type 2 diabetes model for TELO?

Telomir‑Zn reduced HOMA-IR values from about 10–12 to approximately 3 after 14 days, indicating marked insulin resistance improvement. According to the publication, effects were dose-dependent and consistent at Days 7 and 14, alongside better fasting glucose, glucose clearance and fasting insulin.

What does the new Telomir-Zn publication mean for Telomir Pharmaceuticals’ (TELO) triple-negative breast cancer program?

The publication supports Telomir‑Zn’s metal-homeostasis mechanism, which is also targeted in Telomir’s triple-negative breast cancer program. According to the company, these metabolic findings provide additional peer-reviewed validation for biological pathways underlying its FDA-cleared Phase 1/2 TELO-001 TNBC trial.

How does Telomir-Zn’s metal-homeostasis mechanism relate to metabolic disease and cancer for TELO?

Telomir‑Zn was designed to modulate intracellular iron and zinc balance, influencing iron-dependent pathways. According to Telomir, dysregulated metal homeostasis contributes to oxidative stress, mitochondrial dysfunction and altered signaling in both metabolic disease and cancer, supporting its platform across distinct indications.

Is Telomir-Zn already approved to treat Type 2 diabetes or cancer for Telomir Pharmaceuticals (TELO)?

Telomir‑Zn is described as part of a clinical-stage program with an FDA-cleared IND for a Phase 1/2 TNBC trial, not as an approved therapy. According to the company, current diabetes results are preclinical in a zebrafish model, supporting further investigation.

What key metabolic improvements were observed with Telomir-Zn in the preclinical Type 2 diabetes model for TELO?

Telomir‑Zn produced dose-dependent improvements in fasting glucose, oral glucose tolerance, fasting insulin and HOMA-IR in diet-induced diabetic zebrafish. According to the publication, fasting glucose neared control levels and HOMA-IR dropped to about 3 after 14 days of treatment.