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Telomir Pharmaceuticals Reports Peer-Reviewed Publication of Telomir-Zn Data Demonstrating Improved Survival and Multiple Endpoint Benefits in Wilson's Disease

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Telomir Pharmaceuticals (NASDAQ:TELO) announced a peer-reviewed preclinical study of Telomir-Zn in a Wilson's disease zebrafish model, published in Advances in Redox Research. Telomir-Zn showed dose-dependent survival improvement, reduced hepatic copper, better liver biomarkers, improved locomotor function, less tissue degeneration, and multiple oxidative-stress pathway benefits.

The data represent the first peer-reviewed Wilson's disease publication for Telomir-Zn and, according to Telomir, support its broader mechanistic approach, including its lead Triple-Negative Breast Cancer program cleared by the FDA for IND in April 2026.

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Positive

  • Preclinical Wilson's model showed dose-dependent survival improvement with Telomir-Zn
  • Multiple endpoints improved: hepatic copper burden, liver biomarkers, locomotor function, tissue degeneration
  • Data demonstrate reduced copper-induced oxidative stress and attenuated intracellular calcium dysregulation
  • First peer-reviewed Wilson's disease publication strengthens Telomir-Zn scientific package
  • Findings support mechanistic relevance for Telomir's Triple-Negative Breast Cancer program with FDA IND clearance

Negative

  • None.

News Market Reaction – TELO

+7.38%
4 alerts
+7.38% Session close to close
+4.7% Peak Tracked
-3.1% Trough Tracked
$92.16M Market Cap
0.4x Rel. Volume

In the May 20 session, TELO gained 7.38%, reflecting a notable positive market reaction. Argus tracked a peak move of +4.7% during that session. Argus tracked a trough of -3.1% from its starting point during tracking. Our momentum scanner triggered 4 alerts that day, indicating moderate trading interest and price volatility.

Data tracked by StockTitan Argus on the day of publication.

Market Context

The stock moved +7.4% in the session following this news. A strong positive reaction aligns with Tel...
Analysis

The stock moved +7.4% in the session following this news. A strong positive reaction aligns with Telomir’s pattern of meaningful moves on scientific and regulatory milestones, as seen after prior TNBC and IND updates. However, shares still traded about 60.65% below the 52‑week high and below the $1.37 200‑day MA, highlighting an early-stage risk profile. Investors have previously shown both alignment and divergence with good news, so enthusiasm has at times faded after initial spikes.

Historical Context

5 past events · Latest: Apr 30 (Positive)
Pattern 5 events
Date Event Sentiment 24h Move Catalyst
Apr 30 IND clearance Positive -3.5% FDA cleared Telomir‑Zn IND for advanced or metastatic TNBC.
Apr 24 Acquisition closed Positive +1.4% Closed TELI acquisition, consolidating global rights to Telomir‑Zn.
Mar 31 IND submission Positive +14.0% Submitted IND to FDA for Telomir‑Zn in advanced/metastatic TNBC.
Feb 17 Preclinical efficacy Positive +5.5% Reported broad TNBC tumor cell mortality in preclinical models.
Feb 05 Mechanism data Positive -3.5% Released new data on metal modulation and epigenetic mechanisms.

24h Move is the share-price change in the day after each event; other market factors may also have contributed.

Pattern Detected

Positive R&D and regulatory news has often led to favorable moves, but there are notable instances where upbeat updates were followed by short-term declines.

Recent Company History

Over the past few months, Telomir has advanced Telomir‑Zn from mechanistic preclinical work toward the clinic. Key steps included new epigenetic and metal‑modulation data on Feb 5 and broad TNBC cell‑mortality results on Feb 17. The company then submitted an IND on Mar 31, completed the TELI acquisition on Apr 24, and received FDA IND clearance on Apr 30. This publication adds further preclinical validation around oxidative stress and metal homeostasis.

Key Terms

reactive oxygen species (ROS), mitochondrial dysfunction, ast, bilirubin, +2 more
6 terms
reactive oxygen species (ROS) medical
"Reduction of copper-induced reactive oxygen species (ROS) amplification"
Reactive oxygen species (ROS) are highly reactive molecules produced naturally by cells that can damage DNA, proteins and cell structures when present in excess—think of them like biological 'rust' that wears down tissues. For investors, ROS matter because they are linked to many diseases, influence how drugs work or fail, and serve as safety signals or biomarkers in clinical trials, so levels of ROS can affect a therapy’s development, regulatory review and commercial prospects.
mitochondrial dysfunction medical
"leads to progressive oxidative stress, mitochondrial dysfunction, inflammation"
Mitochondrial dysfunction is when the cell’s energy factories, called mitochondria, fail to make enough energy for cells to work properly, which can cause tissue damage and contribute to many diseases. For investors, it matters because therapies, tests, or devices aimed at fixing or detecting this problem can create market opportunities or clinical risk; think of it as a failing car battery that drives demand for repairs, replacements, and related medical products.
ast medical
"Reduction of liver injury biomarkers, including ALT, AST, and bilirubin"
AST (aspartate aminotransferase) is an enzyme found mainly in the liver and heart that leaks into the blood when those organs are injured; doctors measure blood AST levels to detect and monitor tissue damage. For investors, elevated AST in clinical trial data or executive health disclosures can be an early warning of drug safety problems, regulatory scrutiny, treatment delays, or unexpected costs—think of it as a dashboard light signaling potential trouble under the hood.
bilirubin medical
"Reduction of liver injury biomarkers, including ALT, AST, and bilirubin"
A yellowish substance produced when the body breaks down old red blood cells and normally cleaned out by the liver and sent into the digestive tract. Elevated bilirubin is a simple marker that a liver or blood problem exists — like seeing smoke that suggests a possible fire — and can influence diagnosis, safety signals, clinical trial results and regulatory decisions about drugs or tests related to liver function.
histopathological medical
"Reduction of hepatorenal histopathological degeneration"
Relating to the microscopic study of diseased or treated tissue, histopathological describes observations made by examining thin slices of tissue under a microscope to detect changes in cells, inflammation, scarring or abnormal growth. Investors care because these detailed tissue-level findings serve as key evidence of a medical product’s safety and effectiveness—like inspecting fabric fibers to judge whether a repair worked or caused damage—and can strongly influence clinical results, regulatory approval and company value.
triple-negative breast cancer medical
"lead program in Triple-Negative Breast Cancer, for which the Company received"
Triple-negative breast cancer is a type of breast cancer that lacks three common markers used to identify and treat the disease effectively. Because it doesn’t respond to some targeted therapies, it can be more difficult to treat and may have a more aggressive progression. This impacts the development of new treatments and can influence the outlook for healthcare companies involved in cancer research and pharmaceuticals.

AI-generated analysis. How Rhea-AI works. Not financial advice.

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Study Published in Advances in Redox Research Demonstrated Dose-Dependent Survival Improvement, Reduced Hepatic Copper Burden, Improved Liver Biomarkers, and Reduced Tissue Degeneration in a Preclinical Wilson's Disease Model.

MIAMI, FL / ACCESS Newswire / May 20, 2026 / Telomir Pharmaceuticals, Inc. (NASDAQ:TELO) ("Telomir" or the "Company"), a clinical-stage biotechnology company developing small-molecule therapeutics targeting epigenetic and metabolic drivers of cancer and age-related disease, today announced the peer-reviewed publication of preclinical data showing that Telomir-Zn produced dose-dependent survival improvement across multiple endpoints in a Wilson's disease model, supporting the biological activity of Telomir-Zn in Wilson's disease. The study, titled "Intracellular copper redox modulation disrupts ROS-Ca²⁺ amplification in an ATP7B-deficient zebrafish model of Wilson's disease," was published in Advances in Redox Research.

The publication reports that Telomir-Zn produced dose-dependent improvements across multiple endpoints in the preclinical Wilson's Disease model, including reduced copper-associated oxidative stress, reduced hepatic copper burden, improved liver injury biomarkers, attenuated intracellular calcium dysregulation, improved locomotor function, reduced tissue degeneration, and enhanced survival.

The publication is available online at:
Advances in Redox Research Publication

Wilson's disease is a rare genetic disorder caused by mutation in the ATP7B gene, resulting in impaired copper excretion and toxic copper accumulation in the liver, brain, and other organs. The condition leads to progressive oxidative stress, mitochondrial dysfunction, inflammation, and multi-organ tissue injury. Current standard-of-care treatments, primarily copper chelation agents, address systemic copper levels but do not directly target the downstream oxidative and mitochondrial injury pathways associated with disease progression.

According to the publication, Telomir-Zn demonstrated improvements across cellular and in vivo endpoints in the Wilson's disease model, including:

  • Reduction of copper-induced reactive oxygen species (ROS) amplification

  • Attenuation of intracellular calcium dysregulation associated with oxidative stress signaling

  • Preservation of metabolic viability under copper and peroxide challenge

  • Reduction of hepatic copper accumulation

  • Improvement in locomotor and neuromotor performance

  • Reduction of liver injury biomarkers, including ALT, AST, and bilirubin

  • Reduction of hepatorenal histopathological degeneration

  • Improved survival in a dose-dependent manner in the Wilson's disease model

The authors concluded that targeted modulation of labile intracellular copper pools disrupted ROS-Ca²⁺ feedback amplification, mitigated mitochondrial-associated tissue injury associated with copper overload, and produced dose-dependent improvements in survival.

This publication represents Telomir-Zn's first peer-reviewed publication in Wilson's disease and adds independently validated preclinical data across biochemical, functional, histological, and survival endpoints to the Company's scientific package. The findings further support the mechanistic relevance of Telomir-Zn's approach across pathways involving oxidative stress and dysregulated metal homeostasis, including its lead program in Triple-Negative Breast Cancer, for which the Company received FDA IND clearance in April 2026.

"This study demonstrated that modulation of intracellular copper-driven redox activity can disrupt ROS-Ca²⁺ amplification cascades associated with copper toxicity and mitochondrial injury," said Dr. Itzchak Angel, Chief Scientific Advisor of Telomir Pharmaceuticals and corresponding author of the publication.

Dr. Angel continued, "The findings support intracellular metal homeostasis and redox regulation as potentially important biological pathways across diseases involving oxidative stress, mitochondrial dysfunction, and epigenetic dysregulation, including oncology."

"This peer-reviewed publication is an important scientific milestone for Telomir-Zn. The data demonstrated meaningful improvements across a comprehensive set of endpoints in the Wilson's disease model, and we believe the findings are consistent with the broader mechanistic profile of Telomir-Zn as we advance our lead program in Triple-Negative Breast Cancer toward Phase 1/2 clinical initiation," said Erez Aminov, CEO of Telomir Pharmaceuticals.

Telomir Pharmaceuticals will continue evaluating the Wilson's disease program and potential regulatory considerations alongside advancement of its lead TNBC program toward Phase 1/2 clinical initiation.

About Telomir Pharmaceuticals

Telomir Pharmaceuticals, Inc. (NASDAQ:TELO) is a clinical-stage biotechnology company developing small-molecule therapeutics targeting epigenetic and metabolic pathways implicated in cancer, aging, and degenerative disease. The Company's lead program, Telomir-1 (Telomir-Zn), is designed to modulate intracellular metal homeostasis and epigenetic regulation and has received IND clearance from the U.S. Food and Drug Administration for a Phase 1/2 clinical trial in Triple-Negative Breast Cancer. For more information, please visit https://telomirpharma.com/.

Forward-Looking Statements

This press release contains "forward-looking statements" within the meaning of the Private Securities Litigation Reform Act of 1995. These forward-looking statements generally can be identified by the use of words such as "anticipate," "expect," "plan," "can," "could," "would," "may," "will," "believe," "estimate," "forecast," "goal," "project," "guidance," "potential," "intend," "seek," "target" and other words of similar meaning, although not all forward-looking statements include these words. Forward-looking statements may include, but are not limited to, statements regarding the therapeutic potential, mechanism of action, development plans, regulatory pathway, safety profile, clinical utility, market opportunity, and future development of Telomir-1 (Telomir-Zn) and the Company's other product candidates. These forward-looking statements are based on current expectations, estimates, forecasts, and projections, as well as management's beliefs and assumptions, and are subject to significant risks and uncertainties that could cause actual results to differ materially from those expressed or implied by such statements. These risks and uncertainties include, among others, risks related to preclinical and clinical development, the ability to obtain regulatory approvals, the outcome of future studies, reliance on third parties, intellectual property protection, financing needs, market conditions, and the other risks identified in the Company's under the heading "Risk Factors" contained in the Company's Annual Report on Form 10-K and the Company's other filings with the U.S. Securities and Exchange Commission ("SEC"). Forward-looking statements contained in this press release speak only as of the date hereof, and the Company undertakes no obligation to update or revise such statements, whether as a result of new information, future events, or otherwise, except as required by applicable law.

We caution investors not to place undue reliance on the forward-looking statements contained in this press release. You are encouraged to read our filings with the SEC, available at the SEC website, and in the "Investors" section of our website, for a discussion of these and other risks and uncertainties.

Contact Information

Krystina Quintana
Email: info@telomirpharma.com
Phone: (786) 396-6723

SOURCE: Telomir Pharmaceuticals, Inc



View the original press release on ACCESS Newswire

FAQ

What did Telomir Pharmaceuticals (NASDAQ:TELO) announce about Telomir-Zn on May 20, 2026?

Telomir Pharmaceuticals reported peer-reviewed preclinical data showing Telomir-Zn improved survival and multiple disease endpoints in a Wilson's disease zebrafish model. According to Telomir, benefits included reduced hepatic copper, better liver biomarkers, improved locomotor performance, less tissue degeneration and dose-dependent survival gains.

How did Telomir-Zn affect survival in the Wilson's disease model for TELO?

Telomir-Zn improved survival in a dose-dependent manner in an ATP7B-deficient zebrafish Wilson's disease model. According to Telomir, survival benefits were accompanied by reduced copper-induced oxidative stress, attenuation of intracellular calcium dysregulation, and mitigation of mitochondrial-associated tissue injury linked to copper overload.

What key endpoints improved with Telomir-Zn in the Wilson's disease preclinical study for TELO?

Telomir-Zn was associated with reduced hepatic copper accumulation, improved liver injury biomarkers, and better locomotor and neuromotor performance. According to Telomir, the study also showed reduced hepatorenal histopathological degeneration, lower copper-induced reactive oxygen species amplification, and preserved metabolic viability under copper and peroxide challenge.

How does the Telomir-Zn Wilson's disease publication support Telomir Pharmaceuticals' broader pipeline (NASDAQ:TELO)?

The publication adds independently validated preclinical data across biochemical, functional, histological, and survival endpoints for Telomir-Zn. According to Telomir, these findings support intracellular metal homeostasis and redox regulation pathways relevant to diseases with oxidative stress, including its lead Triple-Negative Breast Cancer program cleared for FDA IND.

What mechanisms did Telomir-Zn target in the Wilson's disease zebrafish model for TELO?

Telomir-Zn focused on modulating labile intracellular copper pools and copper-driven redox activity. According to Telomir, this disrupted ROS-Ca²⁺ feedback amplification, attenuated intracellular calcium dysregulation, mitigated mitochondrial-associated tissue injury, and led to improvements in multiple Wilson's disease-related endpoints, including survival.

Why is the Telomir-Zn Wilson's disease study important for Telomir Pharmaceuticals investors?

The study marks Telomir-Zn's first peer-reviewed Wilson's disease publication, strengthening its scientific foundation in metal homeostasis and oxidative stress. According to Telomir, these preclinical results complement its oncology strategy as it advances the Telomir-Zn-based Triple-Negative Breast Cancer program toward planned Phase 1/2 clinical initiation.